1[1]Pan Z J, Hulst M, Feller S, et al. Equally high prevalence of infection with CagA-positive Helicobacter pylori in Chinese patients with peptic ulcer disease and those with chronic gastritis-associated dyspepsia[J]. J Clin Microbiol, 1997, 35:1344-1347.
2[2]Figura M. Helicobacter pylori factors involved in the development of gastroduodenal mucosal damage and ulceration[J]. J Clin Gastroenterol, 1997, 25(Suppl):149-163.
3[3]Covacci A,Censini S,Bugnoli M, et al. Molecular characterization of the 128-Kda immunodominant antigen of Helicobacter pylori associated with cytotoxicity and duodenal ulcer[J]. Proc Natl Acad Sci USA, 1993, 90: 5791-5795.
4[5]Figueiredo C, Quint W, Nouhan N, et al. Assessment of Helicobacter pylori VacA and CagA Genotypes and Host Serological Response[J]. J Clin Microbiol, 2001, 39:1339-1344.
5[6]Tomb J F, White O,Kerlavage A R, et al. The complete genome sequence of the gastric pathogen Helicobacter pylori[J]. Nature, 1997, 388(6642):539-547.
6[7]Odenbreit S,Puls J,Sedlmaiter B, et al. Translocation of Helicobacter pylori CagA into gastric epithelial cells by type IV secretion[J]. Science, 2000, 287:1497-1500.
