1Akhter SA, Skaer CA, et al.Restoration of β-adrenergic signaling in failing cardiac ventricular myocytes viaadenoviral-mediated gene transfer. PNAS, 1997,94:12100～12105
2Rockman HA, Chien KR, et al. Expression of a β-AR kinase 1 inhibitor prevents thedevelopment of myocardial failure in gene-targeted mice. PNAS, 1998,95:7000～7005
3Manh DL, Kent RL, et al. Adrenergic effects on the biology of the adult mammaliancardiocyte. Circulation, 1992,85:790～804
4Du XJ, Autelitano DJ, et al. β2-AR overexpression exacerbates development of heartfailure after aortic stenosis. Circulation, 2000,101:71～77
5Heather R Cross, Charles Steenbergen, et al. Overexpression of the cardiac β2-adenergicreceptor and expression of a β-AR kinase 1 inhibitor both increse myocardialcontractility but have differentiao effects on susceptibility to ischemic injury.Circulation Research, 1999,85:1077～1084
6Hans-Jrg Weig, Kal-Ludwig Laugwitz, et al. Enhanced cardiac contractility aftergene transfer of V2 vasopressin receptors in vivo by ultrasound-guided injection ortranscoronary delivery. Circulation, 2000,101:1578～1585
7Arai M, Matsui H, Periasamy M, et al. Sarcoplasmic reticulum gene expression incardiac hypertrophy and heart failure. Circulation Research, 1994,74(4):555～564
8Zarain-Herzberg A, Maclennan DH, Periasamy M. Characterization of rabbit cardiacsarco(endo) plasmic reticulum Ca2＋ ATPase gene. J biol Chem, 1990,265:4670～4677
9Del Monte F, Harding SE, et al. Restoration of contractile function in isolatedcardiomyocyte from failing human heart by gene transfer of SERCA2a. Circulation,1999,1020:2308～2311
10Miyamoto MI, del Monte F, et al. Adenoviral gene transfer of SERCA2a improves leftventricular function in aortic-banded rats intransition to heart failure. PNAS,2000,97:793～798
