1Belle EV, Tio FO, Chen DH, et al.Passivation of metallic stents after arterial gene transfer of phVEGF165 inhibits thrombusformation and intimal thickening. JACC, 1997, 29:1371～1379
2Asahara T, Bauters C, Pastore C, et al. Local delivery of vascular endothelialgrowth factor accelerates reendothelization and attenuates intimal hyperplasia inbailoon-injured rat carotid artery. Circulation, 1995, 91:2793～2801
3Roopri JK, Rucklidge GJ, Panou C, et al. The effects of exogenous growth factors onmatrix metalloproteinase secretion by human brain tumor cells. Br J Cancer, 2000, 82:52～55
4Ameniya T, Sasamma H, Mifune M, et al. Vascular endothelial growth factor activatesMAP kinade and enhances collagen synthesis in human mesangial cells. Kidney Int, 1999,56:2055～2063
5Den LS, George CJ, Roubin GS, et al. Corrective use of Gianturco-Roubin flex stentsafter percutaneous transluminal coronary angioplasty. J Am Coll Cardiol, 1997, 29:934
6Monnink SHJ, Netherlands G, Boven V, et al. Do we know the long-term effects ofstent implantation on coronary endothelial function?Circulation, 1998, 98(supplement Ⅰ)：Ⅰ～189
8Carter AJ, Laird JR, Farb A, et al. Morphologic characteristics of lesion formationand time course of smooth muscle cell proliferation in a porcine proliferative restenosismodel. J Am Coll Cardiol, 1994, 24:1398～1405
9Safian RD, Gelbfish JS, Erny RE, et al. Coronary atherectomy:clinical,angiographic, and histological findings and observations regarding potential mechanisms.Circulation, 1991, 82:69～79
10Burgess TI, Fisher EF, Ross SI, et al. The antoproliferative activity of c-myb andc-myc antisense oligonucleotides in smooth muscle cells is caused by a nonantisensemechanism. Proc Natl Acad Sci USA, 1995, 92:5051～5055
