1[1]Gogelein H,Hartung J,Englert HC.Molecular basis, pharmac ology and physiologi cal role of cardiac K(ATP) channels[J].Cell Physiol Biochem. 1999, 9(4-5): 227 ~241
2[2]Coraboeuf E,Nargeot J. Electrophysiology of human cardiac cells[J].Cardiova sc Res.1993,27:1713~1725
3[3]Wettwer E,Amosg G,Gath J,et al.Transient outward current in human and rat ven tricular myocytes[J].Cardiovasc Res.1993,27:1662~1669
4[4]Guo W, Xu H, London B, et al. Molecular basis of transient outward K+ curre nt diversity in mouse ventricular myocytes[J].J Physiol.1999 ,15:521
5[5]Casis O,Iriarte M,Gallego M,et al. Differences in regional distribution of K +current densities in rat ventricle[J].Life Sci,1998,63:391~400
6[6]Wang Z,Fermini B,Nattel S.Rapid and slow components of delayed rectifier cur rent in human atrial myocytes[J].Cardiovasc Res,1994,28:1540~1546
7[7]Shimoni Y,Clark RB,Giles WR.Role of an inwardly rectifying potassium current in rabbit ventricular action potential[J].J Physiol,1992,448:709~727
8[8]Wickman KD,Iniguwz-Lluhi JA,Davenport PA,et al.Recombinant G-protein βγ-sub u nits activate the muscarinic-gated atrial potassium channel[J]. Nature, 1994, 368:255~257
9[9]Grover GJ, Garlid KD.ATP-Sensitive potassium channels: a review of their card ioprotective pharmacology[J].J Mol Cell Cardiol ,2000 ,32(4): 677~695
10[10]Fujita A; Kurachi Y.Molecular aspects of ATP-sensitive K+ channels in the ca rdiovascular system and K+ channel openers[J].Pharmacol Ther, 2000 ,85(1): 39~53
2Tomaselli GF, Zipes DP. What causes sudden death in heart failure[J]. Cire Res,2004,95(8) : 754-763.
3Coronel R, Wilders R, Verkerk AO, et al. Electrophy siological changes in heart failure and their implications for arrhythmogenesis[J]. Bioehim Biophys Acta,2013,12(12): 2432-2441.
4Zou Y, Akazawa H, Qin Y, et al. Mechanical stress activates angiotensin Ⅱ type 1 receptor without the involvement of angiotensin Ⅱ [J]. Nat Cell Biol, 2004, 6(6) : 499-506.
5Bisping E, Wakula P, Poteser M, et al. Targeting cardiac hypertrophy: toward a causal heart fallure therapy [J]. J Cardiovasc Pharmaeol, 2014, 64(4) :293-305.
6Sharif-Naeini R, Folgering JH, Bichet D, et al. :sensing pressure m the cardiovascular system: Gq-coupled mechanoreceptors and TRP channels[J]. J Mol Cell Cardiol, 2010, 48(1): 83-89.
7Rana OR, Zobel C, Saygili E, etal. A simple device to apply equibiaxial strain to cells cultured on flexible membranes[J]. Am J Physiol Heart Circ Physiol, 2008, 294 ( 1 ): H532-H540.
8Zobel C, Rana OR, Saygili E, et al. Mechanisms of Ca2+ dependent calcineurin activation in mechanical stretch induced hypertrophy[J]. Cardiology, 2007, 107(4): 281- 290.
9Tsuji Y, Opthof T, Kamiya K, et al. Pacing-induced heart failure causes a reduction of delayed rectifier potassium currents along with decreases in calcium and transient outward currents in rabbit ventricle [J]. Cardiovasc Res, 2000, 48(2) : 300-309.
10Tsuji Y, Zicha S, Qi XY, et al. Potassium channel subunit remodeling in rabbits exposed to long-term bradycardia or tachycardia; discrete arrhythmogenic consequences related to differential delayed-rectifier changes[J]. Circulation, 2006, 113(3) : 345- 355.