1[1]Kannel WB, Cupples A, Agostino RB. Sudden death risk in overt coronary heart disease:the framingham study. Am Heart J, 1987,113:799～ 804
2[2]Wichter T, Breithardt G, Borggrefe M.In Cardiac Arrhythmia:Mechanism, Diagnosis, and Management (eds Podrid, P. J. &Kowey,P.R.).1995.1 219～1 238
3[3]Bmgada J, Brugada R, Brugada P. Right bundle branch block,ST segment elevation in leads V1 ～ V3;a marker for sudden death in patients without demonstrable structural heart disease. Circulation, 1997,96:1 151
4[4]Wang Q, Li Z, Shen J et al . Genomic organization of the human SCN5A gene encoding the cardiac sodium channel. Genomics, 1996,34:9 ～ 16
5[5]Rogers JC, Qu Y, Tanada TN et al . Molecular determinants of high affinity binding of α- scorpion toxin and sea anemone toxin in the S3 ～ S4 extracellular loop in domain V of the Na channel a subunit. J Biol Chem, 1996,271: 15 950～ 15 962
6[6]Tung RT, Shen W, Hammil SC et al . Idiopathic ventricular fibrillation in out - of- hospital cardiac arrest survivors.PACE 17,1994.1 405～ 1 411
7[7]Cobb LA. in Hurst's The Heart ( eds Schlant, R. C. & Alexander, R. W) Mcgraw Hill. New York. 1994, 8th edn,947 ～ 957
8[8]Miyazaki T. Autonomic and antiarrhythmic drug modulation of Sr segment elevation in patients with Brugada syndome. J Am Coil Cardiol, 1996,27:1 061 ～ 1 070
9[9]Brugada P,& Brugada J. Further characterization of the syndrome of right bundle branch block, ST. segment elevation and sudden cardiac death. J Cardiovasc Electrdphysiol, 1997, 8:325 ～ 331
