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β-受体阻滞剂对心力衰竭心室重构的影响 被引量:7

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作者 高丽君
出处 《中国综合临床》 北大核心 2002年第1期16-17,共2页 Clinical Medicine of China
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  • 2[2]M ary-Rabine L, Albert A, Pham TD, et al. The relationship of human atrial cellular electrophysiology to clinical function and ultrastructure[J]. Circ Res,1983,52:188-199.
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  • 4[4]Thijssen VLJL, Ausma J, Liu GS, et al. Strnctural changes of atrial myocardium during chronic atrial fibrillation [J]. Cardiovasc Pathol, 2000, 9:17-28.
  • 5[5]Goette A, Staack T, Rocken C, et al. Increased expression of extracellular sig-nal-regulated kinase and angiotensin-converting enzyme in human atria during atrial fibrillation[J]. J Am Coll Gardiol, 2000,35:1669-1677.
  • 6[6]Goette A, Honeycutt C, Langberg JJ, et al. Electrical remodeling in atrial fibrillation. Time course and mechanisms[J]. Circulation, 1996,94:2968-2974.
  • 7[7]Ausma J, Wijffels M, Thone F, et al. Structural changes of atrial myocardium due to sustained atrial fibrillation in the goat[J]. Circulation, 1997,96:3157-3163.
  • 8[8]Ausma J, Wijffels M, van Eys G, et al. Dedifferentiation of atrial cardiomyocytea as a result of chronic atrial fibrillation[J]. Am J Pathol, 1997,151:985-997.
  • 9[9]Ausma J, Lenders M H, Mast F, et al. Time course of structural changes due to atrial fibrillation in the goat[J]. Circulation,1998,98:1683.
  • 10[10]Ausma J, Ramaekers FCS, Allessie MA, et al. Time course of cardiomyocyte dedifferentiation due to atrial fibrillation in a goat model[J]. J Mol Cell Cardiol, 1999,31: A62.

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