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原发性十二指肠鳞状细胞癌临床分析 被引量:3

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出处 《贵州医药》 CAS 2001年第12期1088-1090,共3页 Guizhou Medical Journal
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  • 1陈锐,叶明福,张哉根.小肠原发性鳞状细胞癌一例[J].中华病理学杂志,2004,33(4):387-388. 被引量:5
  • 2杨光华.小肠//杨光华.中华外科病理学.北京:人民卫生出版社,2002:698-699.
  • 3Liu Y, Jiang L, Cai Q, et al. Predominant association of HLA -B 2704 with ankylosing spondy|itis in Chinese Han patients. Tissue An- tigens, 2010,75:61.
  • 4TASC. Genome -wide association study of ankylosing spondylitis i-dentifies non MHC susceptibility loci. Nat Genet,2010 ;42 :123.
  • 5Cui X, Hawari F, Alsaaty S, et al. Identification of ARTS - ] as a novel TNFR1 - binding protein that promotes TNFR1 ectodomain shedding. J Clin Invest, 2002,110:515.
  • 6Cui X, Rouhani FN, Hawari F, et al. Shedding of the type II IL - 1 decoy receptor requires a muhifunctional aminopeptidase, aminopepti- dase regulator of TNF receptor type 1 shedding. J Immunol, 2005, 171:6814.
  • 7Cui X, Rouhani FN, Hawari F, et al. An aminopeptidase, ARTS - 1, is required for interleukin -6 receptor shedding. J Biol Chem, 2003,278:28677.
  • 8Kanaseki T, Blanchard N, Hammer GE, et ah ERAAP synergizes with MHC class I molecules to make the final cut in the antigenic pep- tide precursors in the endoplasmic reticulum. Immunity, 2006,25 : 795.
  • 9Saric T, Chang SC, Hattori A, et al. An IFN - gamma - induced aminopeptidase in the ER, ERAP1, trims precursors to MHC class 1 -presented peptides. Nat Imnmnol, 2002,3 :1169.
  • 10TASC, WTCCC2. Interaction between ERAP1 and HLA -B27 in ankylosing spondylitis implicates peptide handling in the mechanism for HLA -B27 in disease susceptibility. Nat Genet, 2011,45:761.

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