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人卵巢上皮癌组织中MHC表达的研究

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出处 《内蒙古医学院学报》 2001年第3期186-187,共2页 Acta Academiae Medicinae Neimongol
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  • 1Seung S, Urban JL, Schreiber H. A tumor escape variant that has lost one major histocompatibility complex class Ⅰ restriction element induces specific CD 8+T cells to an antigen that no longer serves as a target[J].J Exp Med, 1993;178:933
  • 2Kabawat SE, Bast RCJr, Welch WR, et al. Expression of major histocompatibility antigens and nature of inflammatory cellularinfiltrate in ovarian neoplasms[J]. Int J Cancer , 1983;32 (5):547 ~ 554
  • 3Kono K, Halapi E, Hising C, et al. Mechanisms of esc ape from CD8+T cell clones specific for the her- 2/neu proto- oncogene expressed in ovarian carcinomas: related and unrelated to decreasedMHC class Ⅰ expression [J]. Int J Cancer , 1997;70(1) :112~119
  • 4Bevan MJ. Antigen presentation to cytotoxic T lymphocytes in vivo[J].J Exp Med , 1995;182:639~641
  • 5Rock KL. A new foreign policy:MHC class Ⅰ molecules monitor theoutside world[J]. Immunol Today , 1996; 17 (3):131 -137
  • 6Allarena P, Presti LO, De Bello M, et al. Proliferative response of lymphocytes from cancer patients to autologous tumor cells[J].Immunol Immunother, 1988; 27: 69 ~ 76

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