1Donaldson K. Mechanisms of pneumoconiosis, in Occupational Lung Disease (eds Banks DE, Parker JE), London: Chapman &Hall, 1998:139～160
2Brody AR, Bonner JC, Overrby LH, et al. Interstitial pulmonary macrophages produce ateletderived growth factor that stimulates rat lung fibroblast proliferation in vitro. J Leukocyte Biol, 1992,51: 640～ 648
3Donaldson K, Gilmour PS, MacNee W, et al. Pree radical activity associated with ultrafine titanium dioxide particles and PM10 material. Am J Resp Crit Care Med, 1995,151:A64
4Hill I, Beswick PH, Donaldson K. Differential release of superoxide anion by macrophages treated with long and short fibre amosite asbestos is a consequence of differential affinity for opsonin. Occup Environ Med, 1995,52:92～96
5Piguet PF, Rosen H, Vesin C, et al. Effective treatment of the pulmonary fibrosis elicited in mice by bleomycin with anti-CD-11 antibodies. Am Rev Respir Dis, 1993,147 :435～441
6Warhek DB, Carakostas MC, Bamberger JR, et al. Complement facilitates macrophage hagocytosis of inhaled iron particles but has little effect in mediating silica-induced lung inflammatory and clearance responses. Environ Res, 1991,56 : 186～ 203
7Driscoll KE, Howard BW, Hassenbein DG, et al. Oxidative stress and silica induced increases in nuclear, NF-κB binding activity in alveolar epithelial cells. Am J Resp Crit Care Med, 1995, 151:A712
8Simeonova PP, Luster MI. Iron and reactive oxygen species in the asbestos-induced tumornecrosis factor-alpha response from alveolar macrophages. Am J Respir Cell Mol Biol, 1995,12:676～683
9Lombard-Gillooly K, Hubbard AK. Modulation of silica-induced lung injury by reducing lung non-protein sulfhydryls with buthionine sulphoximine. Toxicol Lett, 1993,305～315
10Lasky JA, Coin PG, Lindroos PM, et al. Chrysotile asbestos stimulates platelet-derived growth factors-AA production by rat lung fibroblasts in vitro: evidence for an autocrine loop. Am J Respir Cell Mol Biol, 1995,12:162～170
