1[1]Bullock G,Ray S,Reed JC,et al.Intracellular me tabolism of Ara-C and resulting DNA fragmentation and apoptosis of human AML HL .60 cells possessing disparate levels of Bcl-2 protein[J].Leukemia,1996,10(4 ): 1731-1740.
2[2]Freund,Rossig,Lanvers,et al.All-trans-retinoic acid increases cytosine arabinoside cytotoxicity in HL.60 human leukemia cells in sp ite of deceased cellular Ara-CTP accumulation[J].Annual oncology,1999,10(3):3 35-338.
4[4]Meinhardt,Roth,Totok,et al.Signaling defect in the act ivation of caspase-3 and PKC delta in human cells is associated with resistance to apoptosis[J].Experimential cell research,1999,247(2):535-542.
5[5]Viana,Kramer,Saunders,et al.Inhibition of protein kin ase C activator-mediated induction of P21C1P1 and P27K1P1 by deoxycytidine anal ogs in human leukemia cells:relationship to apoptosis and differentiation[J].B iochemical Pharmacology,1999,58 (1):121-131.
6[6]Susan P,Whitmant,Trancesca.Protein kinase C activation by 1-D-arabinofuranosylcytosine is antagonistic to stimulation of apoptosis and Bcl-2 down-regulation[J].The journal of biological chemistry,1997,272(38): 2 3481-23484.
7[7]Barbara H,Liebermann DA.The proto-oncogene c-Myc and apo ptosis[J].Oncogene,1998,17(25):3351-3357.
8[8]Chelliah J,Freemerman AJ,Susanna WP,et al.Protentiat ion of Ara-C induced apoptosis by the protein kinase C activator bryostatin 1 i n human leukemia cells (HL.60) involves a process dependent upon c- Myc[J].B iochemical Pharmacology,1997,54:563-573.
9[9]Kim CN,Wang XD.Overexertion of bcl-xl inhibits Ara-C-in duced mitochondrial loss of cytochrome c and other perturbations that activate t he molecular cascade of apoptosis[J].Cancer Research,1997,57:3115-3120.
10[10]Marina K,Tari AM,Zeev E,et al. Loposomal Bcl-2 antisense oligonucle otides enhance proliferation,sensitize acute myeloid leukemia to cytosine-arabi noside,and induce apoptosis independent of other antiapoptotic proteins[J].Blo od,2000,95(12):3929-3938.