1Renolleau S,Aggoun-Zouaoui D,Ben-Ari Y,et al.A model of transient unilateral focal ischemia with reperfusion in the P7 neonatal rat: morphological changes indicative of apoptosis[].Stroke.1998
2Nunez G,Benedict MA,Hu Y,et al.Caspases: the proteases of the apoptotic pathway[].Oncegene.1998
3Nakajima W,Ishida A,Lange MS,et al.Apoptosis has a prolonged role in the neurodegeneration after hypoxic ischemia in the newborn rat[].The Journal of Neuroscience.2000
4Cheng Y,Deshmukh M,D’Costa A,et al.Caspase inhibitor afford neuroprotection with delayed administration in a rat modal of neonatal hypoxic-ischemic brain injury[].The Journal of Clinical Investigation.1998
5Vanderluit JL,Mcphall LT,Fermandes KJL,et al.Caspase-3 is activiated following axotomy of neonatal facial motoneurons and caspase-3 gene deletion delays axotomy-induced cell death in rodents[].European Journal of Neuroscience.2000
6Wang X,Karlsson JO,Zhu C,et al.Caspase-3 activation after neonatal rat cerebral hypoxia-ischemia[].Biology of the Neonate.2001
7Nath R,Scott M,Nadimpalli R,et al.Activation of apoptosislinked caspase(s) in NMDA-injured brains in neonatal rats[].Neurochemistry International.2000
8Schulz JB,Weller M,Moskowitz MA.Caspases as treatment targets in stroke and neurodegenerative diseases[].Annals of Neurology.1999
9Srinivasula SM,Fernandes-Alnemri T,Zangrilli J,et al.The Ced-3/interleukin 1βconverting enzyme-like homolog Mch6 and the lamin-cleaving enzyme Mch2αare substrates for the apoptotic mediator CPP32[].Journal of Biological Chemistry.1996
10Scott RJ,Hegyi L.Cell death in perinatal hypoxic-ischaemic brain injury[].Neuropathology and Applied Neurobiology.1997
