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AMPA受体与缺血性脑损伤 被引量:1

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摘要 α 氨基 3 羟基 4异恶唑 丙酸 (AMPA)受体介导的兴奋毒性在缺血性脑损伤的发生机制中占有重要地位。
出处 《国外医学(脑血管疾病分册)》 2002年第2期137-139,共3页 Foreign Medical Sciences Cerebrovascular Diseases
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同被引文献5

  • 1Koike K,Tsukada S,Tsuzuki K,et a1.Regulation of kinetic properties of GluR2 AMPA receptor channels by alternative splicing[J].J Neurosci,2000,20(6):2166
  • 2Ozawa S,Ka fiya H,Tsuzuki K.Glutamate receptors in the mammalian central nervous system[J].J Prog Neurobiol,1998,54(5):581
  • 3Weiss JH,Sensi SL.Ca2+-Zn2+ permeable AMPA or kainite receptors:possible key factors in selective neurodegenration[J].Trends Neurosci,2000,23 (8):365
  • 4Steiner P,Sarria JC,Glauser L,et al.Modulation of receptor cycling by neuron enriched endosomal protein of 21 kD[J].J Cell Biol,2002,157(7):1197
  • 5Muranyi M,Fujioka M,Qingping H,et al.Diabetes activates cell death pathway after transient focal cerebral ischemia[J].Diabetes,2003,52:481

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