视网膜缺血损伤的分子生物学机制研究进展被引量：2

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摘要视网膜缺血损伤时 ,因细胞外谷氨酸浓度增加、细胞内Ca2 + 浓度增加 ,从而引起一系列病理生理反应。兴奋性氨基酸可能是视网膜缺血病变过程的触发者。

作者刘静坤马挺雷云洁张德秀

机构地区西安市红会医院眼科西安市第一医院西安交通大学第一医院眼科

出处《国外医学（生理病理科学与临床分册）》 2002年第2期145-146,共2页 Foreign Medical Sciences(Pathophysiology and Clinical Medicine)

关键词视网膜病理学视网膜损伤炎症热休克蛋白类

参考文献15

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