核因子-κB与急性肺损伤关系的研究进展
被引量：1
摘要
核因子(NF)-κB活化不仅促进单核巨噬细胞、中性粒细胞(PMN)表达多种促炎性细因子和粘附分子等,还可能延缓PMN的凋亡,因而与急性肺损伤的关系密切。抑制NF-κB活化可能为ALI的治疗提供了一条新的途径。
出处
《国外医学（呼吸系统分册）》
2002年第2期63-65,69,共4页
Section of Respiratory System Foreign Medical Sciences
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