2[2]Cummins TR, Howe JR, Waxman SG. Slow closed-state inactivation: A novel mechanism underlying ramp current in cells expressing the hNE/PN1 sodium channel. J Neueoscience, 1998,18:9607～9619.
3[3]Akopian AN, Sivilotti L, Wood JN, A tetrodotoxin-resistant voltage -gated sodium channel expressed by sensory neurons. J Biol chem,1996,271:5953～5956.
4[4]Dib - Hajj SD, Tyrrell L, Black JA, et al. NaN a novel voltage-gated Na channel is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy . Proc Natl Acad Sci USA, 1998,95: 8963～8969.
5[5]Felts PA, Black JA, Dib - Hajj SD, et al, NaG: a sodium channelslike mRNA shared by Schwann cells and other neural crest derivatives.Glia, 1997,21:269～277.
6[6]Brock JA, McLachian EM, Belmonte C. Tetrodotoxin-resistant impulses in single nociceptor nerve terminals in guinea-pig cornes. J Physiol, 1998,512:211～217.
7[7]Rizzo MA, Successful treatment of painful traumatic mononeuropathy with carbamazepine: insights into a possible molecular pain mechanism. J neurosci, 1997,36:103～106.
8[8]Zhang JM, Donnelly DF, Song XJ,et al. Axotomy increases the excitability of dorsal root ganglion cells with unmyelian tedaxons . J Netrophysiol, 1997,78: 2790～2794.
9[9]Waxman SG,Kocsis JK, Black JA.Type Ⅲ sodium channel mRNA is expressed in embryonic but not adult spinal sensory neyrons , and is reexpressed following axtomy,J Neurophysiol, 1994,72:466～471.
10[10]Dib - Hajj SD, Black JA, Felts P, et al. Down-regulation of transcripts for Na channel α-SNS in spinal sensory neurons following axotomy. Proc Natl Acad Sci USA, 1996,93:14950～14954.
