1[1]Ledeen RW, Wu GS, Lu ZH, et al. The role of GMl and other gangliosides in neuronal differentiation: overview and new finding. Ann N Y Acad Sci 1998, 845: 161-175.
2[2]Silva RH, Bergamo M, Vital BMBF, et al. Effects of neonatal GM1 administration on the discrimination avoidance behavior of adult rats. Ann N Y Acad Sci 1998, 845: 45-53.
3[3]Ando S, Tanaka Y, Waki H, et al. Gangliosides and sialycholesterol as of synaotic functions. Ann N Y Acad Sci 1998, 845:232-239.
4[4]Schengnmd CL, Mummert CM. Exogenous glanglisides: How do they cross the blood-brain barrier and how do they inhibit cell proliferation? Ann N Y Acad Sci 1998, 845:279-283.
5[5]Frontczak-Baniewicz M, Gadasmki R, Barskov 1, et al. Beneficial effects of GM1 ganglioside on photochemically-induced microvascular injury in cerebral cortex and hypophysis in rat.Exp Toxicol Pathol, 2000, 52(2): 111-118.
6[6]Simon RP, Chen J, Graham SH. GM1 ganglioside treatment of focal ischemia: a dose-response and microdialvsis study. J Pharmacol Exp Ther 1993, 265(1): 24-29.
7[7]Krajnc D, Wemlinger TA, Neff NH, et d. Neonatal hypoxia:early neurotransmitter responses and the consequences of treatment with GM1 ganglioside. J Pharmacol Exp Ther 1994, 271(3): 1299-1306.
8[8]Garofalo L, Cuello A. Nerve growth factor and the monosialoganglioside GM 1: analogous and different in vivo effects on biochemical, morphological, and behavioral paramenters of adult cortically lesioned rats. Exp Neurol 1994, 125: 195-217.
9[9]Rabin S J, Mocchetti J. GM1 ganglioside activaes the high-affmity nerve growth factor receptor TekA. J Neuroch 1995, 65:347-354.
10[10]D'Mello SR, Galli C, Ciotti T, et al. Induction of apoptosis in cerebellar granule neurons by low potassium: inhibition of death by insulin-like growth factor I and cAMP. Proc Natl Acad Sci USA 1993, 90: 10989-10993.
