1Andres V, Pello OM,Silvestre-Roig C. Macrophage proliferation and apoptosis in atherosclerosis[ J]. Curt Opin Lipidol, 2012, 23 (5) :429 -438.
2Moore KJ, Tabas I. Macrophages in the pathogenesis of atheroscle- rosis[J]. Cell, 2011, 145(3) :341 -355.
3Davignon J, Ganz P. Role of endothelial dysfunction in atheroscle- rosis[ J ]. Circulation, 2004, 109 (23 suppl 1 ) :27 - 32.
4Mitra S, Deshmukh A, Sachdeva R, et al. Oxidized low-density lipoprotein and atherosclerosis implications in antioxidant therapy [J]. Am JMedSci, 2011, 342(2) :135-142.
5Jiang X, Yang Z, Chandrakala AN, et al. Oxidized low density lipoproteins-do we know enough about them? [ J]. Cardiovasc Drugs Ther, 2011, 25(5):367-377.
6Ling S, Nheu L, Komesaroff PA. Cell adhesion molecules as phar- maceutical target in atheroscleresis [ J]. Mini Bey Med Chem, 2012, 12(2) :175 -183.
7Tuttolomondo A, Di Raimondo D, Pecoraro R, et al. Atheroscle- rosis as an inflammatory disease[ J]. Curr Pharm Des, 2012, 18 (28) :4 266 -4 288.
8Zeruecke A, Weber C. Chemokines in atheroscleresis: proceed- ings resumed [ J ]. Arterioscler Thromb Vasc Biol, 2014, 34 (4) : 742 - 750.
9Podgrahinska S, Kamaln O, Mayer L, et al. Inflamed lymphatic endothelium suppresses dendritic cell maturation and function via Mac-1/ICAM-l-dependent mechanism [ J ]. J Immunol, 2009, 183(3) :1 767-1 779.
10Tacke F, Alvarez D, Kaplan TJ, et al. Monocyte subsets differ- entially employ CCR2, CCR5, and CX3CR1 to accumulate with- in atheresclerotic plaques [ J ]. J Clin Invest, 2007, 117 ( 1 ) : 185 - 194.