1Misonon H,Maho MK,Yasuo Iet al.oxidative stress induces intracellular accumulation of amyloid beta-protein in human neuroblastonma cells[J].Biochem,2000(39):6951-6959.
2Yang Y,Quitschke WW,Brewer GJ.Upreglulation of amyloid precursor protein gene promoter in rat primary hippocampal neurons by phorbol esrer,IL-1 and retinoic acid,but not by reactive oxygen species[J].Brain Res,1998,60(1):40-49.
4Dela Monte SM,et al.Mitochondrial DNA damages as a mechanism of loss in Alzheimer's disease[J].Lab Invest,2000,80(2):1325-1335.
5Patrizia Mecocci,Usha MacGarvey,Flint Beal.Oxidative damage to mitochondrial DNA is increased in Alzheimer's disease[J].Neurochem,1994.
6Lovell,M.A;Ehmann,W.D :Butler,S.M: et al.Elevated thiobarbituric acid-reactive substances and antioxidant enzyme activity in the brain in Alzheimer's disease[J].Neurology,1995 (45):1594-1601.
7Zsolt Radak,Takao Kaneko,Shoichi Tabara et al.The effect of exercise training on oxidative damage of lipids,proteins,and DNA in rat skeletal muscle:evidence for beneficial outcomes[J].Free Radic.Biol.Med,1999(27):69-74.
8Zsolt Radak,Hae Y chung,Erika koltai,et al.Exercise,oxidative stress and hormesis[J].Ageing res.rev,2008(7):34-42.
9Hyun Seob UM,et al.Exercise training acts as a therapeutic strategy for reduction of the pathogenic phenotypes for Alzheimer's disease in an NSE /APPsw-transgenic model[J].Int.J.Mole.Med,2008(22):529-539.
