摘要
Recent reports demonstrate a role of calmodulin （CaM）-binding DNA-binding proteins （transcription factors [TFs]） as repressors of the plant immune response through their interactions with promoters of a number of target genes. However, the role of Ca2＋ in gene repression has not been tested. Thus, to infer the function of such TFs as repressors of transcription prior to path- ogen attack raises questions about the role of Ca2＋ in this tran- scription repression and subsequently in de-repression upon pathogen attack. Here we discuss open questions and possible mechanisms.
Recent reports demonstrate a role of calmodulin （CaM）-binding DNA-binding proteins （transcription factors [TFs]） as repressors of the plant immune response through their interactions with promoters of a number of target genes. However, the role of Ca2＋ in gene repression has not been tested. Thus, to infer the function of such TFs as repressors of transcription prior to path- ogen attack raises questions about the role of Ca2＋ in this tran- scription repression and subsequently in de-repression upon pathogen attack. Here we discuss open questions and possible mechanisms.
