期刊文献+

不稳定心绞痛长病变冠脉介入治疗的临床观察 被引量:1

Clinical Observation of Unstable Angina Long Lesions of Coronary Intervention
下载PDF
导出
摘要 目的研究不稳定心绞痛长病变冠脉介入治疗的临床观察。方法将我院2013年1月~2014年12月间诊治的110例不稳定心绞痛冠脉造影显示的长病变患者均分为两组,对拒绝行冠脉介入治疗的双号组患者进行单纯药物治疗(对照组),对单号组患者在对照组的基础上采用经皮冠状动脉介入治疗(观察组),对比观察二者的临床治疗效果。结果在总有效率方面,观察组的94.55%高于对照组的80.00%,差异显著,具有统计学意义(P〈0.05)。结论对于不稳定心绞痛长病变患者来讲,采用经皮冠状动脉介入治疗可以显著提高临床治疗效果。 Objective Clinical study of treatment of unstable angina long lesions coronary intervention. Methods Our hospital in January 2013-December 2014 long lesions in patients diagnosis and treatment of 110 patients with unstable angina, coronary angiography were divided into two groups of patients double number of rejected rows coronary intervention were simple drug treatment(control group), on a single number of patients treated by percutaneous coronary intervention(study group), compared to observe the clinical therapeutic effect of both on the basis of the control group. Results In terms of overall efficiency, 94.55% was higher than that of the observation group(80.00%), the difference was significant, with statistical significance(P〈0.05). Conclusion For long lesions in patients with unstable angina is concerned, the use of percutaneous coronary intervention can significantly improve clinical outcomes.
出处 《中国继续医学教育》 2015年第15期94-95,共2页 China Continuing Medical Education
关键词 不稳定心绞痛长病变 冠状动脉介入治疗 药物治疗 临床效果 Unstable angina long lesions Coronary intervention Drug therapy Clinical effect
  • 相关文献

参考文献1

二级参考文献12

  • 1Moore KJ, Tabas I. Macrophages in the pathogenesis of atherosclerosis[J]. Cell, 2011,145 (3): 341.
  • 2Cretney E, Xin A, Shi W, et al. The transcription factors Blimp-1 and IRF4 jointly control the differentiation and function of effector regulatory T cells[J]. Nat Immunol, 2011,12(4) :304-311.
  • 3Keaney JF. Immune modulation of atherosclerosis[J]. Circulation,2011,124(22) :e559-560.
  • 4NNakamura K, Sasaki T, Cheng XW, et al. Statin prevents plaque disruption in apoE-knockout mouse model through pleiotropic effect on acute inflammation[J]. Atherosclero- sis,2009,206(2) :355-361.
  • 5Buja LM, Willerson JT. Role of inflammation in coronary plaque disruption[J]. Circulation, 1994,89(1) :503-505.
  • 6Neri Serneri GG, Prisco D, Martini F, et al. Acute T-cell activation is detectable in unstable angina[J]. Circulation, 1997,95(7): 1806-1812.
  • 7Sakaguchi S, Sakaguchi N, Asano M, et al. Immunologic self-tolerance maintained by activated T cells expressing IL-2 receptor alpha-chains(CD25). Breakdown of a single mechanism of self-tolerance causes various autoimmune diseases[J]. J Immunol, 1995,155(3): 1151-1164.
  • 8Ait-Oufella H, Salomon BL, Potteaux S, et al. Natural regulatory T ceils control the development of atheroscle- rosis in mice[J]. Nat Med,2006,12(2)-178-180.
  • 9van Puijvelde GH, Hauer AD,de Vos P,et al. Induction of oral tolerance to oxidized low-density lipoprotein amel- iorates atheroselerosis [J]. Circulation, 2006, 114 (18): 1968-1976.
  • 10de Boer OJ, van der Wal AC. FOXP3 + regulatory T cells in vulnerable atheroselerotic plagues [J]. Int J Cardiol, 2010,145 (1) :161.

共引文献11

同被引文献6

引证文献1

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部