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hCLP46 increases Smad3 protein stability via inhibiting its ubiquitin-proteasomal degradation

hCLP46 increases Smad3 protein stability via inhibiting its ubiquitin-proteasomal degradation
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摘要 hCLP46 (human CAP10-1ike protein 46 kDa) was initially isolated and identified from human acute myeloid leukemia transformed from myelodysplastic syndrome (MDS-AML) CD34+ cells (Teng et al., 2006) and we demonstrated previ- ously that hCLP46 is abnormally expressed in many hematopoietic malignancies (Wang et al., 2010). hCLP46 (human CAP10-1ike protein 46 kDa) was initially isolated and identified from human acute myeloid leukemia transformed from myelodysplastic syndrome (MDS-AML) CD34+ cells (Teng et al., 2006) and we demonstrated previ- ously that hCLP46 is abnormally expressed in many hematopoietic malignancies (Wang et al., 2010).
出处 《Protein & Cell》 SCIE CAS CSCD 2015年第10期767-770,共4页 蛋白质与细胞(英文版)
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