摘要
Endurance training in the aerobic zone of metabolism promotes a transition from type II to type I muscle fibers, influences the enzyme system of the Krebs cycle, electron transport chain, capillary supply, changes in key metabolic enzymes involved in fatty acid activation, and increased oxygen uptake in skeletal muscle. First of all, the transition process is related to the contractile apparatus. 5＇adenosine monophosphate-activated protein kinase is activated in response to endurance training and related to the metabolic adaptation of skeletal muscle. The peroxisome proliferator-activated receptor isoform δ is an important regulator of skeletal muscle endurance capacity. Resistance training improves muscle contractile function and increases muscle mass as a result of an anabolic and anti catabolic effect in fast-twitch fibers. Resistance training expands the amount of the contractile apparatus in order to enlarge fiber cross-sectional area. A concomitant alteration in proteins phenotype of contractile apparatus and metabolic enzyme levels occurs in accordance with activity-induced changes in the muscle＇s fiber-type profile. Recovery of skeletal muscle mechanical properties depends on the structural and metabolic peculiarities of muscle and the character of training.
