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TWEAK/Fn14在心肌重塑及心衰中的研究进展 被引量:2

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摘要 心肌细胞增殖、心肌病理性重塑、心肌肥大等因素是导致心衰病理进展的主要机制,而炎症因子及其受体在心肌重塑及心衰进展中的作用日益得到关注。近年来研究发现,TWEAK/Fn4轴在心衰炎症反应、纤维化等病理机制中发挥多种功能,作者综述了TWEAK/Fn4轴在心肌重塑及心衰中的研究进展。
出处 《现代医学》 2015年第11期1459-1462,共4页 Modern Medical Journal
基金 湖北省武汉市医药卫生项目(WX10C15)
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参考文献27

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二级参考文献37

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