摘要
急性脑梗死缺血及再灌注损伤机制复杂,丁苯酞作为一种新的神经保护药物可以阻断缺血及再灌注损伤的多个环节,发挥抗脑缺血作用,现已在临床广泛应用。本文就丁苯酞的具体作用机制做一综述。
The mechanism of ischemia and reperfusion injury in acute cerebral infarction is extremely complex.Dl-3-n-butylphthalide,as a new neuroprotective agent which has been widely used in clinic,could play the role of antagonizing cerebral ischemia via blocking multiple links in ischemia and reperfusion injury.In this paper,we review the specific mechanism of dl-3-n-butylphthalide.
出处
《临床荟萃》
CAS
2016年第2期182-186,共5页
Clinical Focus
关键词
脑梗死
丁苯酞
再灌注损伤
brain infarction
dl-3-n-butylphthalide
reperfusion injury
