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人免疫缺陷病毒1感染非特异性免疫研究进展

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摘要 在急性人免疫缺陷病毒1(HIV-1)感染时,病毒的病原体相关分子模式被感染细胞的病原体识别受体所识别,从而触发信号级联反应,启动非特异性抗病毒机制,发挥抗病毒作用。HIV-1感染非特异免疫涉及病原体感知、病毒限制性因子、自然杀伤(NK)细胞的抗病毒作用以及病毒逃逸策略等多个方面。
出处 《山东医药》 CAS 北大核心 2016年第21期95-97,共3页 Shandong Medical Journal
基金 北京市科技计划项目(D131100005313005)
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