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环境性吸烟导致的肺癌生物标记物水平变化及口岸控烟现状

Change of Lung Cancer Biomarkers Level Caused by Environmental Tobacco Smoking and Recent Tobacco Control Situation at Port
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摘要 环境性吸烟可导致肺癌生物标记物水平的变化,通过监测检测NNK、可替宁、CYP1A1基因等的变化,可早期识别肺癌。烟草导致的环境性吸烟和直接吸烟危害,使人罹患癌症、肺部疾病、心血管疾病的几率大增。越来越多国家加入《烟草控制框架公约》,推进了包括口岸公共场所在内的公共场所控烟工作的开展。 Environmental tobacco smoking can cause changes of lung cancer biomarkers level. Lung cancer in early stage can be identified through monitoring and detecting the changes of NNK, cotinine, CYP1A1 gene and others. Harms caused by environmental tobacco smoking and direct tobacco smoking lead to a dramatic rising in the prevalence of cancers, lung and cardiovascular diseases. More and more countries join in the Framework Convention on Tobacco Control, which promotes the tobacco control in public places including the port ones.
出处 《口岸卫生控制》 2016年第6期55-60,共6页 Port Health Control
关键词 环境性吸烟 肺癌 生物标记物 控烟 Environmental tobacco smoking Lung cancer Biomarker Tobacco control
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