摘要
蔗糖非发酵1(SNF1)相关蛋白激酶家族(SnRKs)是植物胁迫响应过程中的一类关键蛋白激酶。在响应生物胁迫时,SnRKs可通过参与活性氧和水杨酸介导的信号转导途径,增强植物对生物侵害的耐受性。在响应非生物胁迫时,SnRKs通过脱落酸(ABA)介导的信号通路,增强植株对干旱、盐碱和高温等的耐受性;且通过独立于ABA的信号通路,SnRKs可调控胞内能量状态,维持离子平衡。该文总结了SnRKs蛋白激酶作为胁迫信号通路中的主要调节因子的最新研究进展,并展望了未来的研究方向。
SnRKs （sucrose nonfermenting-l-related protein kinases） are key protein kinases in stress responses, in response to biotic stresses, they are involved in reactive oxygen species and salicylic acid-mediated signaling transduction pathways to enhance the plant tolerance to biological stress. In response to abiotic stresses, SnRKs enhance the plant tolerance to drought, salinity and high temperatures by intracellular signaling mediated by abscisic acid （ABA）, regulate cellular energy homeostasis and maintain ion balance by ABA-independent signaling. SnRKs are the main regulators in stress signaling in recent research and we give a brief outlook for future study.
出处
《植物学报》
CAS
CSCD
北大核心
2017年第3期346-357,共12页
Chinese Bulletin of Botany
基金
国家自然科学基金(No.31571585,No.31371554,No.30871459)
江苏省农业科学院基本科研业务专项(No.ZX(16)2002)
