期刊文献+

腹腔镜肝癌切除术对原发性肝癌患者术中出血量及术后康复的影响 被引量:8

Effect of Laparoscopic Hepatocellular Carcinoma Resection on Intraoperative Blood Loss and Postoperative Rehabilitation in Patients With Primary Liver Cancer
下载PDF
导出
摘要 目的探讨腹腔镜肝癌切除术对原发性肝癌患者术中出血量及术后康复的影响。方法选取我院原发性肝癌患者82例,根据术式不同分为观察组与对照组,各41例。对照组行开放式肝癌切除术治疗,观察组行腹腔镜肝癌切除术治疗。对比两组手术前后肝功能指标[丙氨酸氨基转移酶(ALT)、天冬氨酸氨基转移酶(AST)、总胆红素(TBIL)]以及两组手术效果、术后康复情况比较。结果观察组术中出血量、手术时间、术后进食时间及肛门排气时间均少于对照组,差异有统计学意义(P<0.05);观察组术后ALT、AST、TBIL低于对照组,差异有统计学意义(P<0.05)。讨论腹腔镜肝癌切除术治疗原发性肝癌患者,可有效改善肝功能指标,降低术中出血量,缩短进食时间。 Objective To investigate the effect of laparoscopic hepatocellular carcinoma resection on intraoperative blood loss and postoperative rehabilitation in patients with primary liver cancer. Methods 82 patients with primary hepatocellular carcinoma in our hospital were divided into observation group and control group according to different surgical procedures, 41 cases in each group. The control group was treated with open hepatectomy and the observation group underwent laparoscopic hepatocellular carcinoma resection. Liver function index [alanine aminotransferase (ALT), aspartate aminotransferase (AST), total bilirubin (TBIL)] and surgical results, postoperative rehabilitation were compared between the two groups before and after operation. Results The intraoperative blood loss, operation time, postoperative eating time and anal exhaust time in the observation group were lower than in the control group, the difference was statistically significant (P 〈 0.05); the ALT, AST and TBIL levels in the observation group were lower than those in the control group after operation(P 〈 0.05). Conclusion Laparoscopic hepatocellular carcinoma resection of primary hepatocellular carcinoma can effectively improve the liver function index, reduce the intraoperative blood loss and shorten the eating time.
作者 王继军
出处 《中国卫生标准管理》 2017年第12期28-30,共3页 China Health Standard Management
关键词 腹腔镜肝癌切除术 原发性肝癌 术中出血量 laparoscopic hepatocellular carcinoma resection primary liver cancer intraoperative blood loss
  • 相关文献

参考文献8

二级参考文献100

共引文献211

同被引文献97

引证文献8

二级引证文献49

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部