1Lee J, Taneja V, Vassallo R. Cigarette smoking and inflamma- tion: cellular and molecular mechanisms[J]. J Dent Res, 2012, 91 (2) : 142-149.
2Lubin JH, Blot WJ, Berrino F, et al. Modifying risk of developing lung cancer by changing habits of cigarette smoking[ J ]. Br Med J ( Clin Res Ed) , 1984, 288 (6435) : 1953-1956.
3Jindal SK, Aggarwal AN, Chaudhry K, et al. A multicentric study on epidemiology of chronic obstructive pulmonary disease and its relationship with tobacco smoking and environmental tobacco smoke exposure [ J ]. Indian J Chest Dis Allied Sci, 2006, 48 (1) :23-29.
4Teo KK, Ounpuu S, Hawken S, et al. Tobacco use and risk of myocardial infarction in 52 countries in the INTERHEART study: a case-control study[ J]. Lancet, 2006, 368 (9536) :647-658.
5Swan GE, Hodgkin JE, Roby T, et al. Reversibility of airways in- jury over a 12-month period following smoking cessation [ J ]. Chest, 1992, 101(3) :607-612.
6Domagala-Kulawik J, Maskey-Warzechowska M, Hermanowicz- Salamon J,et al. Expression of macrophage surface markers in in- duced sputum of patients with chronic obstructive pulmonary dis- ease[ J]. J Physiol Pharmacol,2006,57 Suppl 4:75-84.
7Stockley RA. Neutrophils and the pathogenesis of COPD [ J ]. Chest, 2002, 121(5 Suppl) :151s-155s.
8Le-Barillec K, Si-Tahar M, Balloy V, et al. Proteolysis of mono- cyte CD14 by human leukocyte elastase inhibits lipopolysaccha- ride-mediated cell activation[ J ]. J Clin Invest, 1999, 103 (7) : 1039-1046.
9Takanashi S, Hasegawa Y, Kanehira Y, et al. Interleukin-10 level in sputum is reduced in bronchial asthma, COPD and in smokers[ J]. Eur Respir J, 1999,14 (2) :309-314.
10Hacievliyagil SS, Mutlu LC, Temel I. Airway inflammatory mark- ers in chronic obstructive pulmonary disease patients and healthy smokers[J]. Niger J Clin Pract, 2013, 16(1 ) :76-81.
