摘要
[目的]随着麦田除草剂的大量使用,许多杂草产生严重抗性,精噁唑禾草灵是防除野燕麦的主要药剂(芳氧苯氧丙酸类,AOPP),而有关野燕麦抗药性的报道却不多,旨在探讨野燕麦(Avena fatua L.)对精噁唑禾草灵的抗性。[方法]采用整株测定法测定了河南24个地区野燕麦种群的茎和根分别对精噁唑禾草灵的敏感度差异。[结果]从野燕麦株高看,对精噁唑禾草灵最不敏感的西华(周口)野燕麦种群是最敏感的襄县(许昌)野燕麦种群的151.730倍,其IC_(50)值分别为643.790、4.243 g a.i./hm^2。而从野燕麦的根长看,对精噁唑禾草灵最不敏感的武陟(焦作)野燕麦种群是最敏感的鄢陵(许昌)野燕麦种群的31.643倍,其IC_(50)值分别为25 117.041、793.751 g a.i./hm^2。[结论]野燕麦的茎和根对精噁唑禾草灵的敏感度存在不一致性,但均表明这些地区野燕麦对精唑禾草灵产生了不同程度的抗性,为野燕麦对除草剂的抗性机理研究提供了理论基础。
[Aims] As the herbicides were used heavily, many weeds have produced serious resistance,metsulfuron-methyl is as a kind of main aryloxyphenoxypropionate herbicide(AOPP) to control wild oats in wheat fields, however, the resistance of wild oats is seldom reported. This paper aims to explore the level of resistance of wild oat(Avena fatua L.) to fenoxaprop-p-ethyl. [Methods] The whole plant assay was conducted to determine susceptibility of 24 populations of wild oat in Henan province to herbicide(fenoxaprop-p-ethyl). [Results] The results indicated that the stalk in Xihua( Zhoukou) was the most unsusceptible, while the stalk in Xiangxian( Xuchang) was the most susceptible. The IC_(50) values were 643.790 and 4.243 g a.i./ha, respectively, and the ratio was 151.730 times. However,the root in Wuzhi(Jiaozuo) was the most unsusceptible while the root in Yanling(Xuchang) was the most susceptible.The IC_(50) were 25 117.041 and 793.751 g a.i./ha, respectively, and the ratio was 31.643 times. [Conclusions] Susceptibility of the stalk and root of wild oats to fenoxaprop-p-ethyl is inconsistent, and stalk is more susceptible than root. But it showed that wild oat of these regionshas developed different levels of resistance to fenoxaprop-p-ethyl, which provides theoretical foundation and basis for mechanism of resistance of the wild oat to herbicides.
出处
《农药》
CAS
CSCD
北大核心
2017年第12期922-925,共4页
Agrochemicals
基金
河南省科技公关(162102110018)
河南科技学院重点项目:豫北麦田节节麦抗药性鉴定及抗性种质资源库构建(14QN024)
河南科技学院高层次人才引进启动项目:麦田野燕麦抗精噁唑禾草灵分子机制研究(208010616003)
关键词
野燕麦
抗药性
除草剂
精噁唑禾草灵
敏感度
wild oat (A venafatua L.)
resistance
herbicide
fenoxaprop-p-ethyl
susceptibility