期刊文献+

排龈线技术治疗牙体楔状缺损充填修复后的悬突、炎症的临床效果研究 被引量:8

Study on the clinical effect of gingival line technique in the treatment of the suspensions and inflammation after filling and repairing of the dental wedge defect
下载PDF
导出
摘要 目的观察牙体楔状缺损充填修复后应用排龈线技术治疗对悬突、炎症的预防效果。方法选取接受充填修复治疗的128例牙体楔状缺损患者临床资料,按治疗方案不同分两组,各64例,对照组行常规充填复合树脂修复,观察组在实行排龈线技术后充填复合树脂修复,比较组间炎症、牙周健康及修复情况。结果两组治疗后的炎症因子含量均比治疗前升高(P<0.05),但观察组治疗后的AST、TNF-α、IL-1β和IL-6均比对照组低(P<0.05);观察组治疗后PLI、BI、PD、AL指标值均比对照组低(P<0.05);观察组随访1年后发现填充体脱落3.13%、边缘不密合1.56%、悬突7.81%均比对照组低(P<0.05)。结论牙体楔状缺损充填修复后应用排龈线技术治疗能有效减轻炎症反应,改善牙周健康,预防悬突,促进修复,效果显著。 Objective To observe the preventive effect of gingival line technique in the treatment of suspension and inflammation. Methods Select filling repair treatment of clinical data of 128 patients with tooth wedge-shaped defects, according to the different treatment is divided into two groups, 64 cases, control group routine filling composite resin restoration, observation group in the row of gum line after filling composite resin restoration technology, comparison between groups of inflammation, periodontal health and repair. Results The levels of inflammatory factors in both groups were higher than those before treatment(P〈0.05), but the AST, TNF-α, IL-1β and IL-6 after treatment were lower than those in the control group(P〈0.05). After treatment, PLI, BI, PD and AL index values were lower than those in the control group(P〈0.05). After follow-up for 1 year,the observation group found that the filling body was shedding 3.13%, the edge was not close to the margin of 1.56%, and the suspension was 7.81%,which was lower than the control group(P〈 0.05). Conclusion It is effective to reduce the inflammatory response, improve periodontal health, prevent overhanging process and promote repair.
作者 江进波 Jiang Jinbo(Jingshan People’s Hospital,Jingmen,Hubei, 431899,China)
机构地区 京山县人民医院
出处 《当代医学》 2018年第15期45-47,共3页 Contemporary Medicine
关键词 牙体楔状缺损 充填修复 排龈线技术 悬突 炎症 Dental wedge defect Filling and repairing Technology of gum line Suspension process Inflammation
  • 相关文献

参考文献13

二级参考文献78

共引文献59

同被引文献45

引证文献8

二级引证文献9

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部