出血性脑梗死发病机理及治疗探讨

Study on Onset Mechanism and Treatment of Hemorrhagic Infarction

目的探索出血性脑梗死发病机理及治疗的具体对策。方法选取该院2015年1月—2017年6月收治的出血性脑梗死患者共100例进行了临床研究,对患者血压、神经缺损情况、颈动脉粥样硬化、患者的血脂和高敏C-反应蛋白变化情况等进行了对比研究。结果治疗1个月后SBP、DBP的数值分别为(132.2±2.2)mmol/L、80.2±1.0)mmol/L;治疗1个月后SBP、DBP的数值分别为(133.4±1.5)mmol/L、(83.4±5.6)mmol/L;治疗1个月后SBP、DBP的数值分别为(133.7±4.4)mmol/L、(82.1±6.6)mmol/L。SCL-90评分治疗1月后数值为(64.32±5.2)分;治疗6个月后数值为(78.71±2.5)分。神经功能缺损评分治疗1月后数值为(12.56±2.3)分;治疗6个月后数值为(11.35±5.5)分。MMSE评分治疗1月后数值为(25.21±1.5)分;治疗6个月后数值为(26.22±2.5)分。MBI评分治疗1月后数值为(64.40±3.3)分;治疗6个月后数值为(75.05±5.5)分。TG治疗后数值为1.53±0.26)mmol/L、TC治疗后数值为(1.53±0.26)mmol/L、LDL-C治疗后数值为(1.53±0.26)mmol/L、HDL-C治疗后数值为1.53±0.26)mmol/L、Hs-CRP治疗后数值为(1.53±0.26)mg/L。差异有统计学意义(P<0.05)。结论出血性脑梗死的发病机制是多元化的,必须动态观察,及时了解发病机制,进行对症治疗。

Objective To explore the specific strategies of onset mechanism and treatment of hemorrhagic infarction.Methods 100 cases of patients with hemorrhagic infarction admitted and treated in our hospital from January 2015 to June 2017 were selected for clinical research, and the blood pressure, nerve defect situation, carotid artherosclerosis,blood liquid and changes of high sensitive C reactive protein were compared and researched. Results After 1-month treatment, the SBP and DBP values were respectively（132.2±2.2）mm Hg,（80.2±1.0）mm Hg; after 1-month treatment,the SBP and DBP values were respectively（133.4±1.5）mm Hg,（83.4±5.6）mm Hg, after 1-month treatment, the SBP and DBP values were respectively（133.7±4.4）mm Hg,（82.1±6.6）mm Hg. After 1-month treatment, the SCL-90 score was（64.32±5.2）points; after six-month treatment, the SCL-90 score was（78.71±2.5）points, after 1-month treatment and six-month treatment, the nerve function defect scores were respectively（12.56±2.3）points and（11.35±5.5）points, and after 1-month treatment and six-month treatment, the MMSE scores were respectively（25.21±1.5）points and（26.22±2.5）points, after 1-month treatment and six-month treatment, the MBI scores were respectively（64.40±3.3）points and（75.05 ±5.5）points, after treatment, the TG, TC, LDL-C, HDL-C, Hs-CRP were respectively（1.53 ±0.26）mmol/L,（1.53 ±0.26）mmol/L,（1.53 ±0.26）mmol/L,（1.53 ±0.26）mmol/L,（1.53 ±0.26）mg/L, and the differences were statistically significant（P〈0.05）. Conclusion The onset mechanism of hemorrhagic infarction is multiple, and we must conduct the dynamic observation, know the onset mechanism in time and conduct the systematic treatment.