摘要
Hemidystonia is frequently due to an underlying structural lesion in the basa l ganglia and thalamus. It has been suggested that a preserved corticospinal tra ct may be required for hemidystonia to manifest. We provide the first report of a patient who presented with rapid- onset hemidystonia precipitated by an acute pontine infarct demonstrated on diffusion- weighted magnetic resonance imaging . Acute dysregulation of pallidal efferents to the pedunculopontine and/or ponti ne afferents to the thalamus may precipitate hemidystonia.
Hemidystonia is frequently due to an underlying structural lesion in the basa l ganglia and thalamus. It has been suggested that a preserved corticospinal tra ct may be required for hemidystonia to manifest. We provide the first report of a patient who presented with rapid- onset hemidystonia precipitated by an acute pontine infarct demonstrated on diffusion- weighted magnetic resonance imaging . Acute dysregulation of pallidal efferents to the pedunculopontine and/or ponti ne afferents to the thalamus may precipitate hemidystonia.
出处
《世界核心医学期刊文摘(神经病学分册)》
2005年第10期41-41,共1页
Digest of the World Core Medical Journals:Clinical Neurology