摘要
离体大鼠心脏灌流实验表明,三丙酮胺(TAA)能改善缺血心肌的收缩功能,降低缺血心叽中过氧化产物丙二醛(MDA)含量,保护超氧化物歧化酶(SOD)的活性,提高胞浆肌酸磷酸激酶(CPK)含量,并有效地抑制CaCl_2诱导的血小板聚集作用.
Triacetonamine(TAA)could improve the constructile function of heart mus-cles of acute anemia and decrease the content of malodialdehyde (MDA) in the heart muscles. TAA protects the activitis of superoxide dismutase(SOD) and creatine phos-phate kinase (CPK). It also inhibits CaCl2 induced agglomeration of thromocyte.
出处
《中山大学学报论丛》
1994年第6期4-6,共3页
Supplement to the Journal of Sun Yatsen University
关键词
三丙酮胺
丙二醛
超氧比物歧化酶
肌酸磷酸激酶
血小板聚集
triacetomamine
malodialdehyde
superoxide dismutase
creatine phosphate kinase
agglomeration of thromocyte
