摘要
炎症反应是卒中后神经细胞死亡的重要病理过程。白细胞介素(interleukin, IL)-33/ST2系统参与了缺血性卒中的炎症反应过程,且被证实是一种保护因素。文章对IL-33/ST2系统在缺血性卒中后免疫炎症调节中的作用及机制进行了综述。
Inflammation response is an important pathological process of neuronal cell death after stroke. Interleukin (IL)-33/ST2 system is involved in the inflammatory response process of ischemic stroke and has been proved to be a protective factor. This article reviews the role and mechanism of IL-33/ST2 system in the regulation of immune inflammation after ischemic stroke.
作者
胡睿瑶
裴璐璐
刘新静
许予明
宋波
Hu Ruiyao;Pei Lulu;Liu Xinjing;Xu Yuming;Song Bo(Department of Neurology, the Fust Affiliated Hospital of Zhengzhou University、Zhengzhou 450052, China)
出处
《国际脑血管病杂志》
2018年第12期939-943,共5页
International Journal of Cerebrovascular Diseases
基金
国家自然科学基金(8157051749,8160051522).
