摘要
芒果炭疽病是芒果生长期和采后贮藏期的主要病害之一,严重影响芒果的产量和品质。本文从芒果炭疽病的症状、病原菌分类鉴定、生物学特性、侵染特性及致病机理等方面进行综述;针对芒果炭疽病症状复杂、我国芒果炭疽病病原菌未获详细而系统的研究,指出全面了解我国芒果炭疽病的病原菌种类及优势种群,明确我国芒果炭疽菌的致病力强弱,有助于研究该病的发生流行规律,为抗病材料的选育和抗病品种在田间的合理布局提供参考。提出对我国芒果炭疽菌的抗药性进行系统监测分析,可防止或减缓芒果炭疽菌对杀菌剂抗药性的产生,从而有效减少化学农药的用量,为研究芒果炭疽病绿色防控新策略、新方法和新药剂奠定基础。
Mango anthracnose is one of the main diseases of mango during growth stage and post harvest and storing period that causes seriously affecting the yield and fruits quality.In this paper,the symptom,pathogen classification and identification,biological characteristics,infection features and pathogenesis mechanism of mango anthracnose were reviewed.Aiming at the complexity of the symptoms of mango anthracnose,and the pathogens of mango anthracnose has not been studied in detailed and systematically,infer that comprehensively understand the categories of anthracnose pathogens and their dominant population,and make certain the strength and weakness of virulence of anthracnose in China,will conducive to study the occurrence and epidemic law of the disease,and provide references for the breeding of disease resistant material and reasonable layout of the disease resistant varieties in the field.Put forward the performing of systematic monitoring and analyzing the anthracnose resistance,and may prevent and slacken the inspiring of fungicide resistance against mango anthracnose in China,so as to effectively dampen the dosage of pesticide,and lay the foundation for study on green prevention and control of mango anthracnose new strategies,new methods,and new medicaments.
作者
李其利
卜俊燕
唐利华
黄穗萍
郭堂勋
莫贱友
LI Qi-li;BU Jun-yan;TANG Li-hua;HUANG Sui-ping;GUO Tang-xun;MO Jian-you(Inst.of Plant Protect.,Guangxi Acad.of Agric.Sci.,Guangxi Key Lab.of Biol.for Crop Dis.&Insect Pests,Nanning 530007)
出处
《微生物学杂志》
CAS
CSCD
2020年第1期117-124,共8页
Journal of Microbiology
基金
国家自然科学基金项目(31560526,31600029)
广西留学回国基金重点项目(2016GXNSFCB380004)。
关键词
芒果炭疽菌
分类鉴定
生物学特性
侵染特性
致病机理
mango anthracnose
classification and identification
biological characteristics
infection characteristics
pathogenic mechanism