摘要
Extract The contrary but interrelated processes of axon degeneration and regeneration are the yin and yang of many neurodegenerative conditions. Here we discuss recent evidence for metabolic cross-talk between glia and injured axons regulating these processes. We especially focus on potential bioenergetic mechanisms as to how axon-flanking glia may promote regeneration.
基金
supported by grants from the National Institute of Health(R01NS111024)
the Muscular Dystrophy Association(577844)
Funding provided through the Empire State Development Corporation for Hunter James Kelly Research Institute grant nos.W753 and U446
the Hunter’s Hope Foundation(to BB)。
