2[2]Kurosawa H, Que F G, Roberts L R et al. Hepatocytes in the bile duct-ligated rat express Bcl-2. Am J Physiol,1997,272:1587
3[3]Bonaldo M F, Lennon G, Soares M B. Normalization and subtraction: two approaches to facilitate gene discovery. J Genome Res, 1996,6(9):791
4[4]Arora A S, deGroen P C, Emort Y et al. A cascade of degradative hydrolase activity contributes to hepatocyte necrosis during anoxia. Am J Physiol, 1996,270 (33):238
5[5]Osse T, Olivier R, Monney L et al. Bcl-2 prolongs cell survival after Bax-induced release of cytochrome c. Nature, 1998,391:496
6[6]Mitsuhiro I, Kenichi H, Naoko K et al. Expression of Bcl-2 familial proteins is reduced in small bile duct lesions of primary biliary cirrhosis. Hum Pathol, 2000,31:179
7[7]Maser R S, Antoku K, Scully W J et al. Analysis of the role of conserved cysteine residues in the bcl-2 oncoprotein. Biochem Biophys Res Commun, 2000,277(1):171
8[8]Lebedeva I, Rando R, Ojwang J et al. Bcl-xL in prostate cancer cells: effects of overexpression and down-regulation on chemosensitivity. Cancer Res, 2000, 60 (21):6052
9[9]Korsmeyer S J, Marx J, Debatin K M et al. Bcl-2 initiates a new category of oncogenes: regulators of cell death. Blood, 1992,80:879
10[10]Parker J E, Mufti G J, Rasool F et al. The role of apoptosis, proliferation, and the Bcl-2-related proteins in the myelodysplastic syndromes and acute myeloid leukemia secondary to MDS. Blood, 2000,96(12):3932