摘要
After more than 50 years since Fisher and Adams’original description in 1964,1 transient global amnesia(TGA)is still a puzzling syndrome.Over time,several mechanisms have been proposed for TGA pathogenesis,including venous congestion,cortical spreading depression,seizures and arterial ischaemia.2–4 A sufficient amount of studies in favour and against each of these mechanisms has emerged,fostering uncertainty regarding aetiology as well as prognosis.To this extent,most of the controversy stands on whether TGA represents a risk factor for stroke,or whether this is just as benign as originally thought.
