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伴多器官结构及功能损伤的Loeffler心内膜炎病案1例

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摘要 Loeffler心内膜炎男性较多见,其特征是心肌中嗜酸性粒细胞浸润和脱颗粒,导致炎症、组织损伤、血栓形成,最终心肌纤维化发展为限制性心肌病,出现一侧或两侧心室顺应性急剧下降的急性舒张功能障碍。
出处 《实用老年医学》 CAS 2022年第11期1174-1176,1179,共4页 Practical Geriatrics
基金 国家重点研发计划(2018YFC1707402)。
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  • 1肖志坚,郝玉书,卞寿庚,秘营昌,李建波,陈辉树,钱林生,丁现超,王岩,薛艳萍,赵耀中.8p11骨髓增殖综合征——一例报告及文献复习[J].中华血液学杂志,1996,17(11):566-568. 被引量:10
  • 2Gotlib J, Cools J, Malone JM, et al. The FIP1L1-PDGFRa fusion tyrosine kinase in hypereosinophilic syndrome and chronic eosinophilic leukemia: implications for diagnosis, classification,and management. Blood,2004,103(8) :2879-2891.
  • 3Brito-Babapulle F. The eosinophilias, including the idiopathic hypereosinophilic syndrome. Br J Haematol,2003, 121 (2): 203-223.
  • 4Tefferi A. Modern diagnosis and treatment of primary eosinophilia.Acta Haematol, 2005,114(1):52-60.
  • 5Molecular GJ classification and pathogenesis of eosinophilic disorders: 2005 Update. Acta Haematol,2005,114(1):7-25.
  • 6Roufosse F, Cogan E, Goldman M, et al. The hypereosinophilic syndrome revisiled. Annu Rev Med,2003,54(2):169-184.
  • 7Golub TR, Barker GF, Lovett M, et al. Fusion of PDGF receptor beta to a novel ets-likegene, tel, in chronic myelomonocytic leukemia with t(5; 12) chromosomal translocation. Cell, 1991,77(2) :307-316.
  • 8Pardanani A, Reeder T, Porrata LF, et al. Imatinib therapy for hypereosinophilic syndrome and other eosinophilic disorders. Blood, 2003,101(9): 3391-3397.
  • 9Apperley JF, Gardembas M, Melo JV, et al. Response to imatinib mesylate in patients with chronic myeloproliferative diseases with rearrangements of the platelet-derived growth factor receptor beta. N Engl J Med, 2002.347(7): 481-487.
  • 10Chen J, DeAngelo DJ. Kutok JL, et al. PKC412 inhibits the zinc finger 198-fibroblast growth factor receptor 1 fusion tyrosine kinase and is active in treatment of stem cell myeloproliferative disorder. Proc Natl Acad Sci USA, 2004,101 (40):14479-14484.

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