脑卒中并发脑心综合征的机制及临床特点

The Mechanism and clinical characteristics of stroke complicated with cerebrocardiac syndrome

脑心综合征是指在缺血性卒中、脑出血、蛛网膜下腔出血、急性脑外伤等颅脑损伤因素作用下,导致下丘脑或脑干损伤引起的继发性心脏损害。在临床工作中发现急性脑血管病患者心肌损伤标志物升高、心肌缺血、心律失常等异常高发。随着脑卒中引起心脏损伤研究的深入,为脑心之间交互作用提供新思路,为临床脑心相关并发症的防治提供解决方案。本文概述了脑心综合征,探讨其发病机制,包括神经-体液调节机制、炎症相关机制以及临床相关表现和检测异常,旨在为引起足够的临床重视,达到早期识别、早期处理,减少脑心综合征引起的严重并发症及住院费用。

Brain heart syndrome refers to the secondary heart damage caused by hypothalamus or brain stem injury under the action of brain injury factors such as ischemic stroke,cerebral hemorrhage,subarachnoid hemorrhage and acute brain injury.In clinical work,found that myocardial injury markers increased,myocardial ischemia,arrhythmia and other abnormal high incidence of patients with acute cerebrovascular disease.With the deepening of the research on heart related injury caused by stroke,it will provide a new idea for the interaction between brain and heart,and also provide a scheme for the prevention and treatment of brain heart related complications in clinical work.In this paper,we summarize the brain heart syndrome,discuss its pathogenesis,including neurohumoral regulation mechanism,inflammation related mechanism,clinical manifestations and abnormal detection.The purpose is to cause enough clinical attention after acute stroke,achieve early recognition,early treatment,reduce the serious complications and hospitalization costs caused by cerebral heart syndrome.