Effect of CTRP13 regulates high glucose-induced autophagy dysfunction of primary rat liver sinusoidal endothelial cells through the AMPK/mTOR pathway

Objective To investigate the effect of Clq/tumor necrosis factor-related protein 13 protein (CTRP13) on the autophagy function of primary rat liver sinusoidal endothelial cells (rLSECs) induced by high glucose through AMP-activated protein kinase/mammalian target of rapamycin complex (AMPK/mTOR) pathway.