5KOnigsrainer I, Tiirck MH, Eisner F, et al. The gut is not only the target but a source of inflammatory mediators inhibi- ting gastrointestinal motility during sepsis. Cell Physiol Bio-chem, 2011, 28(4):753-760.
6Sonnier DI, Bailey SR, Schuster RM, et al. Proinflammatory chemokines in the intestinal lumen contribute to intestinal dysfunction during endotoxemia. Shock, 2012, 37(1) : 63-69.
7Rittirsch D, Huber Lang MS, Flierl MA, et al. Immunode- sign of experimental sepsis by cecal ligation and puncture. Nat Protoc, 2009, 4(1):31-36.
8Hietbrink F, Besselink MG, Renooij W, et al. Systemic in- flammation increases intestinal permeability during experi- mental human endotoxemia. Shock, 2009, 32(4):374-378.
9Yajima S, Morisaki H, Serita R, et al. Tumor necrosis fac- tor-alpha mediates hyperglycemia augmented gut harrier dys- function in endotoxemia. Crit Care Med, 2009, 37 (3): 1024-1030.
10Keshavarzian A, Farhadi A, Forsyth CB, et al. Evidence that chronic alcohol exposure promotes intestinal oxidative stress, intestinal hyperpermeability and endotoxemia prior to development of alcoholic steatohepatitis in rats. J Hepatol, 2009, 50(3):538-547.
