1Kuizenga K, Wierda JM, Kalkman CJ, et al. Biphasic EEG changes in relation to loss of consciousness during induction with thiopental, propofol, etomidate, midazolam or sevoflurane. Br J Anaesth, 2001,86:354-360.
2Tufano R, Palomba R, Lambiase G, et al. The utility of bispectral index monitoring in general anesthesia. Minerva Anestesiol, 2000,66 : 389-393.
3Gajraj RJ, Doi M, Mantzarialis H,et al. Analysis of the EEG bispectrum, auditory evoked potentials and EEG power spectrum during repeated transitions from consciousness to unconsciousness. Br J Anaesth, 1998, 80:46-52.
4Cook DJ, Housmans PR. Mechanism of the negative inotropic effect of propofol in isolated ferret ventricular myocardium. Anesthesiology, 1994,80 : 859-871.
5Buljubasic N, Marijic J, Berczi V, et al. Differecntial effects of etomidate, propofol and midazolam on calcium and potassium channel currents in canine myocardialcells. Anesthesiology, 1996,85 : 1092-1099.
6Gelissen HP, Epema AH, Henning RH, et al. Inotropic effects of propofol, thiopental, midazolam, etomidate and ketamine on isolated human atrial muscle. Anesthesiology, 1996,84:397-403.
7Stowe DF, Bosnjak ZJ, Kampine JP. Comparison of etomidate, ketamine, midazolam, propofol and thiopental on function and metabolism of isolated hearts. Anesth Analg, 1992,74 : 547-558.
8Muzi M,Berens RA,Kampine JP,et al. Venodilation contributes to propofol-mediated hypotention in humans.Anesth Analg, 1992,74 : 877-883.
9Mulier JP,Wouters PF,Van Aken H,et al. Cardiodynamie effects of propofol in comparison with thiopental: assessment with a transesophageal eehoeardiographie approach. Anesth Analg, 1991,72 : 28-35.
