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多形性日光疹氯苯吩嗪治疗前后患者血清某些细胞因子的测定 被引量:1

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出处 《中国麻风皮肤病杂志》 北大核心 2003年第1期33-34,共2页 China Journal of Leprosy and Skin Diseases
基金 中华皮肤科学会杨森基金资助项目
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参考文献9

  • 1Morita A, Grewe M, Grether-Bcek S, et al. Induction of proinflammatory cytokines in human epidermoid carcinoma cells by in vitro ultraviolet A1 irradiation. Photochem Photobiol,1997,65(4) :630
  • 2Epstein JH. Polymorphous light eruption.Dermatol clin, 1986,4(2) :243
  • 3Petit-frere C, Clingen PH, Grewe M, et al.Induction of interleukin-6 production by ultraviolet radiation in normal human epidermal keratinocytes and in human keratinocyte cell line is mediated by DNA damage. J Invest Dermatltol, 1998,111(3) :354
  • 4SKOV L, Hansen H, Allen M, et al. Contrasting effects of ultraviolet A1 and ultraviole B exporsure on the induction of tumor necrosis facotr-a in human skin. Br J Dermatol, 1998,138(2):216
  • 5Fujisawa H, Wang B, Kondo S, et al. Costimulation with ultraviolet B and interkeukin-1a dramaticalyy increase tumor necrosis factora production in human dermal fibroblasts. J Interferon Cytokine Res, 1997,17(5) :307
  • 6Danme JV, Opdenakker G, Simpson RJ, et al. Identification of human 26-KD protein, interferon b2(INF-2b) as a B cell hybirdoma/plasmacytoma growth factor induced by interleukin-1 and tumor necrosis factor. J Exp Med,1987 ,165:914
  • 7Larsen CG, Anderson AO, Oppenheim JJ, et al. Production of interleukin-8 by human dermal fibroblasts and keratinocyte-in response to interleukin-1 or tumor necrosis factor. Immunology, 1989,68:31
  • 8Strickland I, Rhodes LE, Flanagan BF, et al. TNF-α and IL-8 are upregulated in the epiedermis of normal human skin after UVB exposure: correlation with neutrophil accumulation and E-selectin expression. J Invest Dermatol, 1997,108( 5 ): 763
  • 9Anderson R, Zeis BM, Anderson IF. Clofazimine-mediated enhancement of reactive. Oxidant production by human phogocytes as a possible therapeutic mechanism. Dermatologica, 1988,176:234

同被引文献13

  • 1邓丹琪,李继梅,谢红,李学平,冒长峙.系统性红斑狼疮、多形性日光疹病人血清中Th_1/Th_2型细胞因子的测定[J].皮肤病与性病,2004,26(4):1-2. 被引量:2
  • 2Sharma L, Basnet A. A clinicoepidemiological study of polymorphic light eruption. Indian Dermatol Venereol Leprol 2008 ; 74( 1 ) : 15 - 17.
  • 3Janssens AS, Pavel S, Ling T, et al. Susceptibility to UVA and UVB provocation does not correlate with disease severity of polymorphic light eruption. Arch Dermatol 2007; 143(5) :599 - 604.
  • 4Epstein JH. Polymorphous light eruption. Photodermatol Photoimmunol Photomed 1997 ; 13 : 89 - 90.
  • 5Palmer RA, Friedmann PS. Ultraviolet radiation causes less immunosuppression in patients with polymorphic light eruption than in controls. J Invest Dermatology 2004; 122 (2) : 291 - 294.
  • 6Seite S, Zucchi H, Moyal D, et al. Alterations in human epidermal langerhans cells by ultraviolet radiation: quantitative and morphological study. Br J Dermatol 2003 ; 148 (2) : 291 - 299.
  • 7Kogen W, van Meurs M, Jongsma M, et al. Differential expression of cytokines in UVB - exposed skin of patients with polymorphous light eruption: correlation with Langerhans cell migration and immunosuppression. Arch Dermatol 2004 ; 140: 295 - 302.
  • 8Janssens AS, Pavel S, Out- Luiting JJ, et al. Normalized UV induction of Langerhans cell depletion and neutrophil infiltrates after artificial UVB hardening of patients with polymorphic light eruption. Br J Dermatology 2005; 152:1268 - 1274.
  • 9Teunissen MB, Piskin G, di Nuzzo S, et al. Ultraviolet B radiation induces a transient appearance of IL - 4^ + neutrophils, which support the development of Th2 responses. J Immunol 2002; 168:3732 - 3739.
  • 10Rhodes LE. Polymorphic light eruption: does a neutrophil defect contribute to the pathogenesis? J Invest Dermatol 2004; 123:13 -15.

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