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甲状腺功能亢进与糖代谢紊乱机制的研究近况 被引量:8

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作者 黎建绿
出处 《广西医学》 CAS 2003年第3期400-402,共3页 Guangxi Medical Journal
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  • 3[3]Kudelska G, Gorski J, Swiatecka J, et al. Effect of exercise on glycogen metabolism in muscles of triiodothyronine-treated rats.Eur J Appl Physical 1996;72(5/6) :496
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  • 5[5]Moller N, Nielsen S, Nyholm B, et al. Glucose tumover, fuel oxidation and forearm substrate exchange in patients with thyroitoxicosis before and after medical treatment. Clin Endocrinal(Oxf)1996;44(4) :453
  • 6[6]Bech K, Damsbo P, Eldrup E, et al. Beta-cell function and glucose and lipid oxidation in Graves' disease. Clin Endocrinal(Oxf) 1996;44(1) :59
  • 7[7]Kemp HF, Hundal HS, Taylor PM. Glucose transport correlates with GLUT2 abundance in rat liver during altered thyroid status.Mol Cell Endocrinal 1997;128(1/2) :97
  • 8[8]Weinatein SP,O'Boyle E,Fisher M,et al. Regulation of GLUT2 glucose transporter expression in liver by thyroid hormone: evidence for hormone regulation of the hepatic glucose transport system. Endocrinology 1994;135(2):649
  • 9[9]Voldstedlund M,Tranun-Jensen J,Handberg A,et al. Quantity of Na Na/K-ATP ase and glucose transporters in the plasma membrane of rat adipocytes is reduced by in vivo triiodothyrome. Eur J Endocrinal 1995;133(5):626
  • 10[10]Bhattacharyya A, wile PG. Diabetic ketocidosis precipitated by thyrotoxicosis. Postgrad Med J 1999;75(3) :291

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