摘要
本文通过对14名心血管病血瘀证患者血液动力学观察,北与非血瘀证患者(n=29)及正常对照组(n=24)比较,表明血瘀证患者的血液动力学改变乃以后负荷加重为特征,而不表现为心脏收缩功能的减退。该改变与血瘀证的血液流变性异常具有相同的意义。与已有的关于心气虛表现为左心收缩功能减退的研究结论相联系,似可认为,由负荷加重,最终累及心脏收缩功能减退为血瘀向气虚血瘀转化的病理生理基础。
In this paper, hemodynamics in 14 cases of cardiovascular diseases with blood-stasis syndrome, 29 cases of the same diseases without blood-stasis syndrome and 24 normal subjects were observed. The results showed that hemodynamic variation in those with blood-stasis syndrome was characterized by the increase of afterload and not the decrease of heart systolic function. Their manifestations appeared with the same signficance of the abnormality of hemorheology in blood-stasis syndrome. Other results indicated that the left ventricular systolic function was decreased in cases with deficiency of heart-qi. So it may be considered that the decreased heart systolic function developed finally due to increased afterload is the pathophysiological basis of qi-dificiency and blood-stasis syndrome which developed from blood-stasis syndrome.
