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试论肾虚痹证(类风湿性关节炎)发育生物学基础 被引量：2

Background of Developmental Biology on Bi Syndrome of the Deficiency of Kidney(Rheumatoid Arthritis)
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摘要 According to traditional Chinese medicine,the kidney promotes the growth and develepment of the body,the deficiency of kidney is a important pathogenesis of Bi syndome (Rheumatoid Arthritis,RA). Modern medicial research shows that RA has abnormality in developmental biology.The paper detects the background of developmental biology on Bi syndrome of the deficiency of kidney. According to traditional Chinese medicine,the kidney promotes the growth and develepment of the body,the deficiency of kidney is a important pathogenesis of Bi syndome (Rheumatoid Arthritis,RA). Modern medicial research shows that RA has abnormality in developmental biology.The paper detects the background of developmental biology on Bi syndrome of the deficiency of kidney.
作者 张浩 吕爱平
机构地区 中国中医研究院基础理论研究所
出处 《中国中医基础医学杂志》 CAS CSCD 2003年第2期8-10,共3页 JOURNAL OF BASIC CHINESE MEDICINE
关键词 肾虚痹证 发育生物学 类风湿性关节炎 中医学 Bi Syndrome Rheumatoid arthritis
分类号 R255.6 [医药卫生—中医内科学]
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参考文献4

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  • 1吕爱平,王安民,曾晓莲.益肾蠲痹丸对大鼠实验性痹证影响的病理学研究[J]中医杂志,1988(06).

共引文献64

同被引文献16

  • 1侯丽萍.对类风湿性关节炎治疗的系统思考[J].山西中医学院学报,2005,6(4):57-58. 被引量：1
  • 2沈自尹.中医虚证参考标准[J].中西医结合杂志,1986,6(10):598-598.
  • 3翟道荡,丁邦友,刘蓉,华雪桂.艾灸关元、中脘和大椎穴调节β-END作用的比较[J].上海针灸杂志,1997,16(1):33-34. 被引量：34
  • 4中华医学会风湿病学分会.类风湿关节炎诊断及治疗指南[J].中华风湿病学杂志,2012,16(2):4-6.
  • 5Edwards JC, Szczepanski L, Szechinski J, et al. Efficacy of B cell-targeted therapy with rituximab in patients with rheumatoid arthritis[J]. N Engl J Med, 2004, 350(25) 2572 -2581.
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  • 8Radstake TR, Franke B, Wenink MH, et al. The functional variant of the inhibitory Fcgamma re- ceptor lib (CD32B) is associated with the rate of radiologic joint damage and dendritic cell function in rheumatoid arthritis [ J ]. Arthritis Rheum, 2006, 54 ( 12 ) :3828 - 3837.
  • 9Neogi T, Aletaha D, Silman A J, et al, The 2010 A- merican College of Rheumatology/European League against Rheumatism classification criteria for rheu- matoid arthritis: phase 2 methodological report[J ].Arthritis Rheum, 2010, 62(9) : 2582 -2591.
  • 10Cataldn D, Aravena O, Sabugo F, et al. B cells from rheumatoid arthritis patients show important alterations in the expression of CD86 and Fc-yR I b, which are modulated by anti-tumor necrosis factor therapy [J]. Arthritis Res Ther, 2010, 12 (2) : 68.

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中国中医基础医学杂志
2003年 第2期

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