热休克蛋白与肿瘤

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摘要热休克蛋白(heat shoch protein,HSP)是在各种理化因素(如热、冷、缺氧、重金属离子、病毒感染等)刺激下,生物细胞内诱导合成的一类高度保守的蛋白质[1].

作者李淑艳张英博王睿

机构地区齐齐哈尔医学院生物化学教研室

出处《医学研究通讯》 2003年第2期54-54,48,共2页 Bulletin of Medical Research

关键词热休克蛋白肿瘤 HSP 理化因素热冷缺氧重金属离子病毒感染

参考文献5

1Chen Y, Robles AI, Martinez LA, et al. Expression of G1 cyclin- dependent kinase, and cyclin - dependent kinase inhibitors in androgen - induced prostate proliferation in castrated rats. Cell Growth Differ, 1996,1571 - 1578.
2Lindquist S, Craing NE. The heat shock proteins. Annu Rev Genet, 1988, 22:631-677.
3Cadyrllino F, Boucher PE, Eichelberg K, Mayhew M, Rothman JE,Houghton AN, Gennain RN. Receptor- mediated uptake of antigen/heat shock protein complexes results in mafor histocompatibility complex class I antigen presentation via two distinct processing pathways. J Exp Med,2000, 191 : 1957- 1964.
4Stangl K, Gunther C, Frank T, Lorenz M. Inhibition of the Ubiquit in-Proteasome Pathway Induces Differential Heat- Shock Protein Response in Cantiomyocytes and Renders Early Cantiac Prietction. Biochem Biophys Res Commun, 2002, 291:542-549.
5Zhang XP, Glaser E. Interaction of plant mitochondrial and chloroplast signal peptides with the Hsp 70 molecular.ular chaperone. Trends Plant Sci,2002, 7:14-21.

医学研究通讯

2003年第2期

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