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颅脑爆炸伤后脑组织兴奋性氨基酸含量变化的实验研究 被引量:3

Experimental study of content changes of excitatory amino acid after craniocerebral ex plosive wound
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出处 《第三军医大学学报》 CAS CSCD 北大核心 2003年第5期461-462,共2页 Journal of Third Military Medical University
基金 全军"十五"指令性课题 ( 0 1L0 6 0 )
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  • 1[1]Hovda D A, Bdcker D P, Katayama Y. Secondary injury and acidosis[J]. J Neurotrauma, 1992,9(Supply 1):S47-60.
  • 2[2]Kawamata T, Katayama Y, Hovda D A, et al. Lactate accumulation following concussive brain injury: the role of ionic fluxes induced by excitatory amino acids[J]. Brain Res, 1995,674(2):196-204.
  • 3[3]Douglas S, Tracy M. Traumatic brain injury and excitatory amino acids. In: Narayan RA, Wilberger J, Povlishock J. Neurotrauma[M]. New York: McGraw-Hill Compames, 1996.1445-1458.
  • 4[4]Takeshi M, Katayama Y, Kawamata T, et al. Mechanisms of excitatory amino acid release in contused brain tissue[J]. J Neurotrauma, 1998,15(9):655-664.
  • 5[5]Lekieffre D, Meldrum B S. The primidine-derivative, BW1003C87, protects CA1 and striatal neurons following transient severe forebrain ischemia in rats: A microdialysis and histological study[J]. Neurosic, 1993,56(1):93-99.

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